Bacteria, saliva, and overall health

http://www.cnn.com/2009/H…ref=mpstoryview

First, Seth Roberts blogs on Oral Health, Heart Disease, and Fermented Foods here:

http://www.blog.sethroberts.net/2009/03/12/oral-health-heart-disease-and-fermented-foods/

A relevant snippet:

Epidemiologic data have shown a statistical association between periodontal disease and coronary heart disease and stroke. In a meta-analysis, the odds ratio increase for CVD in persons with periodontal disease was almost 20%. Poor oral health also seems to be associated with all-cause mortality.

Emphasis added. As I blogged earlier, during my last trip to the dentist I was told my gums were in great shape, better than the previous visit — and the only intentional change since the previous visit was a huge increase (a factor of 50?) in how much fermented food I eat. So perhaps fermented foods improve oral health. A reason to suspect that fermented foods reduce heart disease is that Eskimos, with very low rates of heart disease, eat lots of fermented food. If both these ideas are true — fermented foods improve gum health and reduce heart disease — it would explain the observed correlation between gum disease and heart disease. …

The shift to a diet high in sugar and refined flours has usually happened at the same time as a shift away from traditional diets. In other words, the increase in sugar and flour wasn’t the only change. I suspect there was usually a great reduction in fermented foods at the same time. Maybe the reduction in fermented foods caused the trouble rather than the increase in sugar and flour. The reduction in fermented foods is almost always ignored – for example, by Weston Price and John Yudkin (author of Sweet and Dangerous).

Cross-posting here a comment I made on Seth Robert’s blog post:

I saw a potentially relevant article on saliva and bacteria in CNN recently:

http://www.cnn.com/2009/HEALTH/03/03/saliva.spit.survey/index.html?iref=mpstoryview

A quote:

Since people have different eating habits in different places, you might think an American’s saliva might look a lot different from, say, a South African’s. But a new study published in the journal Genome Research finds that bacteria in saliva may not be as related to environment and diet as you might think.

In fact, researchers found that the human salivary microbiome — that is, the community of bacteria in saliva — does not vary greatly between different geographic locations. That means your saliva is just as different from your neighbor’s as someone’s on the other side of the planet.

Americans in particular have a lot of amylase in their saliva because their diets are full of starch: chips, rice and baked potatoes. But the Pygmies of central Africa, for example, eat mostly game animals, honey and fruit. They have relatively little amylase in their saliva.

Dominy and colleagues found these differences at the genetic level, meaning natural selection has favored large quantities of amylase in populations with starchy diets.

But there is also evidence that amylase levels can rise and fall within an individual’s lifetime. A study on college students in Ghana, who typically eat a lot of meat at the university, found that students who had grown up eating traditional starchy Ghanaian home-cooked meals had lower levels of amylase after attending the school.

Finally, trying to get Stephan of WholeHealthSource hooked up with Seth Roberts as I’m willing to bet there might be some synergies in their research and experimentation on fermentation (particularly as examining the changing diets a la Weston Price’s research).

(H/T Nathan)

Quotes on Sugar, Insulin, Glucose, Aging

Via the IF Life comes a smattering of quotes from Dr. Ron Rosedale on sugar, insulin, glucose, carbohydrates, aging, disease, etc. In short, they are all about the importance of insulin control for your health! Here’s one particularly insightful quote:

We only have one hormone that lowers sugar, and that?s insulin. Its primary use was never to lower sugar. We?ve got a bunch of hormones that raise sugar, cortisone being one and growth hormone another, and epinephrine, and glucagon.

Our primary evolutionary problem was to raise blood sugar to give your brain enough and your nerves enough and primarily red blood cells, which require glucose. So from an evolutionary sense if something is important we have redundant mechanisms. The fact that we only have one hormone that lowers sugar tells us that it was never something important in the past.

Okay, one more:

What they are finding on these major centenarian studies is that there is hardly anything in common among them. They have high cholesterol and low cholesterol, some exercise and some don?t, some smoke, some don?t. Some are nasty as can be and some nice and calm and nice. Some are ornery, but they all [have] low sugar, relatively for their age. They all have low triglycerides for their age.

And they all have relatively low insulin. Insulin is the common denominator in everything I?ve just talked about. They way to treat cardiovascular disease and the way I treated my stepfather, the way I treated the high risk cancer patient, and osteoporosis, high blood pressure, the way to treat virtually all the so-called chronic diseases of aging is to treat insulin itself.

For the full Rosedale lecture, go here.

Caloric Restriction, Red Wine, and Aging

First, a summary:

  • The physiological stress resulting from caloric restriction may extend your life. It could accomplish this by switching resources from reproduction to self-preservation. Alternatively (or additionally), it might accomplish this by helping downregulate insulin, thereby reducing the diseases associated with metabolic syndrome.
  • Resveratrol (Via red wine, for now) might also slow aging via some SIRT-1 mechanism or by somehow signaling to the body that stressful times lay ahead.
  • The stress from exercise3 might slow aging by activating physiological mechanisms of tissue self-preservation, as well.
  • And finally, I can chalk another one up for intermittent fasting, which personal experience says is the easiest way to practice caloric restriction.

The Details

My brother sent me a NYTimes article that talks about resveratrol, a molecule found in red wine, and its possible link to slowing aging. Resveratrol may thwart aging by spurring the production of sirtuins a.k.a. SIRT1 in humans. Per the Times:

[T]he door has now been opened to drugs that exploit an ancient biological survival mechanism, that of switching the body’s resources from fertility to tissue maintenance. The improved tissue maintenance seems to extend life by cutting down on the degenerative diseases of aging.

The reflex can be prompted by a faminelike diet, known as caloric restriction, which extends the life of laboratory rodents by up to 30 percent but is far too hard for most people to keep to and in any case has not been proven to work in humans [See Footnote 1 for discussion on this last sentence].

Whereas the Times article focuses on resveratrol, summarily dismissing caloric restriction, a 2006 article from The Economist does the opposite, going into more detail on the impact of caloric restriction on human aging and a recent study on the matter:

[E]vidence has been accumulating since the 1930s that calorie restriction … extends lifespan and delays the onset of age-related diseases in rats, dogs, fish and monkeys. …

Amid the hype, it is easy to forget that no one has until now shown that calorie restriction works in humans. That omission, however, changed this month, with the publication of the initial results of the first systematic investigation into the matter. This study, known as CALERIE2 (Comprehensive Assessment of Long-term Effects of Reducing Intake of Energy), was sponsored by America’s National Institutes of Health. …

CALERIE suggests the [advantages of caloric restriction] are real. For example, those on restricted diets had lower insulin resistance … and lower levels of low-density lipoprotein [LDL] cholesterol. They showed drops in body temperature and blood-insulin levels?both phenomena that have been seen in long-lived, calorie-restricted animals. They also suffered less oxidative damage to their DNA.

The Economist article goes on to talk about resveratrol and even exercise:

Resveratrol is produced when a vine is under stress?for example, due to dehydration or over-exposure to sunshine. According to Dr Sinclair’s theory, which he calls xenohormesis, animals rely on such botanical stress signals to give them extra information about their own environments, in the same way that the alarm calls of one species warn others of danger. If bad things are happening to plants, he surmises, that is a reason for pre-emptive animal action. Animal bodies thus react to molecules such as resveratrol by activating their own defence mechanisms. These, in turn, protect their cells from stress-related damage.

Xenohormesis is a variation of a more general theory, hormesis … A good example of hormesis is exercise. In theory, this should damage cells because it increases oxygen uptake, and oxidative stress is bad for things like DNA. Of course, exercise is not actually bad for cells?and the reason is that the body activates defence mechanisms which overcompensate for the stress the exercise creates, producing beneficial effects. So, while chronic stress is always bad for you[3], a short period of mild stress followed by a period of recovery can be good.

More on hormesis here.

So what’s the common denominator between exercise, resveratrol, and caloric restriction? Stress. The introduction of acute stress may switch the body’s resources from reproduction to self-preservation4. From an evolutionary perspective, this makes sense. If a band of human beings were facing environmental hardship, such as a famine, their bodies would need to self-preserve until times more suitable for the ultimate biological imperative, reproduction.

Beyond acute stress causing self-preservation and thereby slowing aging, there may be another angle here: insulin. Caloric restriction will reduce insulin loads on the body as entering a fasted state will require the body to switch from glucose-burning (Insulin upregulated) to fat-burning (Insulin downregulated). As I’ve blogged before, chronically high insulin (via diets high in carbohydrates) in the blood is positively correlated to metabolic syndrome or Syndrome X. The connection to slowed aging and lower insulin levels could just be the other side of the same coin, but if nothing else, it’s more support for incorporating some amount of caloric restriction in your life.

Footnotes:

1 I’ve got a question out to the author, Nicholas Wade, as to his last claim regarding caloric restriction and its impact (or lack thereof) on humans. I’m fairly certain that Wade means that the life-extending impact of caloric restriction has yet to be proven for humans, even though the CALERIE study mentioned above would contradict such a claim.

I take serious issue with Wade’s ambiguity here. Most of his article is about resveratrol and sirtuins and their potential link to slowing aging in humans. He goes into detail about a pharmaceutical company that is working on producing a drug that might induce the same effects. That’s fine, but why slam the door on an alternative (caloric restriction) that might slow aging for free? Is it because it’s “far too hard for most people”? How did he determine that?

Update 2008-06-13: Received a response to my email to Nicholas Wade. From part of my email:

Or do you mean that sufficient testing on caloric restriction’s impact on human aging has not occurred, thereby the theory remains unproven?

He indicated that the above interpretation was the one he intended. I will let him know about the CALERIE study.

2 Here is the official CALERIE website.

3 Perhaps therein lies some support for the claim that endurance training is unhealthy as it puts a chronic load of stress on the body.

4 Not surprisingly, Art De Vany mentions as much in passing here:

I practice intermittent caloric deprivation. This is a known enhancer of the immune system. This is pure evolutionary reasoning. During deprivation, the system reallocates resources from reproduction to repair and maintenance. The immune system is part of that adaptation.

More on Coconut Oil

Before I go into the details, here are my big takeways:

  • Cook with coconut oil — try it instead of olive oil for sauteing fish, beef, pork, etc.
  • The medium-length saturated fats in coconut oil are good for “quick energy” because they require little digestion before being quickly absorbed into the body. I cooked with coconut oil at lunch today and was sort-of jazzed all afternoon. Further testing will be required. Addendum: Found this abstract of a study on medium-chain triglycerides: the study showed a 12% rise in basal metabolism on MCTs as compared to only a 4% rise for LCTs.
  • Once again fats win out over carbohydrates. And with even more tasty fats to choose from, its only that much more compelling to jettison the crummy carbs from my diet.

The Details

I picked up some coconut oil from Wal-Mart yesterday. Mind you, it was hardly the uber-natural, ultra-low-processed stuff I should be buying, but I only realized that today. I’ll work on getting the good stuff later; in the meantime, the Louana brand will do just fine.

As curious as I am, after reading the afore-blogged glowing review of oils high in saturated fats, and coconut oil in particular, I had to learn more. Some basic googling led me to this page, which ugliness aside, is pretty informative. Here’s a hearty snippet, as emphasized or edited (…) by me:

Coconut oil is one of the most stable oils you can buy. It does not turn
rancid easily. … coconut oil was one of the foods Weston Price studied in his journeys. He discovered that the coconut was considered, by the local populations, a medicine food. He found that those civilizations that consumed coconut regularly had no knowledge of cancer, heart disease, arthritis, and diabetes.

Let’s take a look at the healing properties of coconut oil:

  • Coconut oil is antiviral, antifungal (kills yeast too) and antibacterial. It attacks and kills viruses that have a lipid (fatty) coating, such as herpes, HIV, hepatitis C, the flu, and mononucleosis. It kills the bacteria that cause pneumonia, sore throats, dental cavities, urinary tract infections, meningitis, gonorrhea, food poisoning, pneumonia, and many, many more bacterial infections. It kills the fungus/yeast infections that cause candida, ringworm, athletes foot, thrush, jock itch, diaper rash and more.
  • Coconut oil is called the “low fat” fat. … It boosts one’s energy and endurance. Many athletes use it blended into their drinks. It also supports thyroid function and increases your metabolism (great if you want to lose weight).
  • Coconut oil improves digestion and absorption of fat soluble vitamins, minerals (especially calcium and magnesium), and amino acids. It improves the body’s use of blood glucose and improves insulin secretion and absorption (great for type II diabetes). In fact, many diabetics (type I and type II) use it to reduce their symptoms. One’s risk of diabetes decreases with regular use of coconuts and coconut oil. And as we already mentioned, cooking with coconut oil does not create any harmful byproducts.
  • Coconut oil helps the body heal and repair faster. It aids and supports immune function, protecting us from a variety of cancers.
  • Coconut oil, contrary to much hubbub, is good for your heart. It keeps our blood platelets from sticking together (and causing dangerous clots). Regular users of coconut oils have a much lower chance of atherosclerosis (clogging of the arteries), arteriosclerosis (hardening of the arteries), and strokes. Coconut oil can lower your blood pressure.
  • Coconut oil is a natural antioxidant. It protects the body from free radical damage and prevents premature aging and degenerative diseases.
  • Finally, coconut oil is the best massage oil on the planet. What it does to your skin, you simply have to witness. It forms a barrier against infections, softens and moisturizes your skin, and prevents wrinkling, sagging, and age spots. It promotes healthy hair and complexion, protects from any damaging UV rays. …


These are some pretty extravagant claims. And unfortunately, they were not footnoted or referenced. A book by Bruce Fife was mentioned (See the nearest match available on Amazon, to the right), which might go into detail on some or all of these claims.

Setting aside some of the more panacea-esque claims, its hard to miss the correlation between consuming coconut oil and preventing metabolic syndrome (A.K.A. “diseases of civilization”), which may be linked to loss of insulin sensitivity or the damage of abundant insulin, which we know is linked to eating carbohydrates. Though I can only speculate as to cause-and-effect, its reasonable to assume that cultures whose diets have a higher percentage of fats in them relative to carboyhdrates are less likely to succumb to the problems associated with insulin (hypertension, diabetes, obesity, cardiovascular disease, etc. — metabolic syndrome).

The bacteria-destroying aspect of coconut oil is intriguing, as well. I understand why saturated fats naturally have a longer shelf-life (Lack of easily broken, carbon=carbon double-bonds), but I can’t help but wonder if another reason coconut oil takes so long to go rancid is some anti-bacterial trait of the oil, itself.

Some extracurricular reading:

Better Health via Intermittent Fasting and a Low-Carb Diet

I’ve been on a low-carb diet now for about two-and-a-half months. This has consisted of eliminating all breads, potatoes, and starchy foods from my diet (Regarding fruit, I pass on bananas with only the occasional apple while still eating berries and other colorful fruits). Furthermore, it has involved intermittent fasting. Through this diet and with minimal exercise, I have reduced my body fat percentage from around 20% to around 10% representing a loss of some 15+ lbs of fat and the gain of a handful of pounds of lean tissue (Weight change from approx. 182 to 168).

This weight loss was the easiest, most satisfying change in my health and body composition I’ve ever experienced.

As this post is extensive, here is the general breakdown:

  • Intro and Intermittent Fasting
  • Low-carb diet
  • Implementation and Conclusion
  • Additional Reading
  • Footnotes

Here are the details:

Intermittent Fasting

Yes, you read correctly, I used the “f word”: fasting. And I can read your mind:

  • What? You starved yourself?
  • Is that for religious reasons?
  • Yeah, that’s called anorexia!

No, no and no. And if I missed any others, no to them, too.

I first got turned onto Intermittent Fasting (IF) via Richard Nikoley over at Free the Animal who had been practicing Art Devaney’s evofitness sans fasting for about a year. Upon trying IF, Richard immediately noticed results in the form of both significant weight loss as well as change in appetite.

Richard’s successes seemed interesting enough from a distance (Fasting? Fascinating but not for me!), but what catalyzed a personal trial in IF and low-carb came after watching a lecture1 by science writer Gary Taubes, author of Good Calories, Bad Calories (“GCBC”).

The big takeaway from the referenced lecture was that insulin, not overeating or under-exercising, is the chief culprit in why people become fat. Since eating carbohydrates causes the pancreas to secrete insulin, there is a direct correlation between carbohydrate consumption and insulin secretion. Ipso facto, the argument is made that eating carbs makes you fatGCBC.

Could it really be that simple? Dieting and working out had failed to reduce my weight effectively — it certainly seemed like something else was affecting my weight. Richard’s success and Taubes’ conclusion sufficiently piqued my interest. Admittedly without knowing all the data, I chose to do an IF, low-carb experiement.

What exactly is intermittent fasting? Intermittent fasting is choosing not to eat for a set period of time, which unlike your daily sleeping fast, is a sustained break from regularly occurring feeding. Translated into some bright line rules, I’d define an intermittent fast as going at least sixteen hours without eating (call that a “short” fast) or going for as long as 30 – 36 hours or three meals (“long fast”). As defined here, a fast requires there be no dietary caloric load on your body.

Fasting for longer than 36 hours likely will not only result in diminishing marginal returns, but it could also start messing with your metabolism. Most obviously, all fasts must be broken by eating.

Completing one to two long fasts per week so long as they are separated by eating a few meals can result in some drastic health benefits while causing no harmful effects on metabolism so long as you are completing some form of regular high-intensity exercise2.

What happens when you fast? Some interesting biological things, apparently. For one, the body moves to mobilize fat stores from the adipose tissue (the fat under your skin) to consume the fat as energy. It accomplishes this fat mobilization as a natural extension of reducing insulin concentrations in the blood as well as an increase in fat mobilizing hormones such as adrenaline, noradrenaline and growth hormone. Interestingly enough, growth hormone is also released to conserve protein from catabolism. Protecting the proteins in your body during a fast is important because your body needs its lean tissues to survive (be they muscle or organ).

A benefit of fasting (for your mind) is that it alters your perspective on eating. At the end of a 30 hour fast, you want to eat something good for your body. You don’t want to gnosh on some french fries, slam a sugary soft drink and eat a bowl of ice cream. Even more, via abstaining from food even when it is available for consumption, you are putting your mind in control of your behavior. Taking back a bit of control over your life is an empowering feeling that leads to improved self-image and confidence.

Biologically, whether fasted or fed, your body is going to take measures to maintain adequate energy flows to demanding muscles and organs. Your body gets this energy from dietary sources or from storage within the body. The key here is that your body is always working to strike an appropriate balance (homeostasis) given the current demands. Were it not managing this process, both eating too much or eating too little would result in your untimely demise!

The ability of our bodies to regulate energy during times of feast or famine is evolutionary engineering. It is reasonable to posit that homo sapiens have only recently lived in such abundance that they could expect to eat food throughout the day, three times a day or more. Go back 50,000 years and you’ve still got mostly the same genetic footprint for human beings, but an entirely different supply of food. These were times when “foraging for food” meant more than a run to the pantry. In other words, our genetics have been engineered to allow us to go without food for longer than ten hours without resulting in our bodies failing. For a tidbit more on the evolutionary aspects of intermittent fasting, see footnote 7.

The bottom line: by reducing insulin in the body and up-regulating fat mobilizing and protein-protecting hormones, IF naturally turns your body into a fat burning machine. During a fast, your body will use whatever energy necessary, which will be similar to the amount of energy required were you eating normally. Over a long fast, if your body requires 2,500 calories, you could expect the majority of that energy to come from fat stores. Thus, the combination of IF with exericse is an effective way to reduce fat stores and work towards a leaner body composition.

Now, the astute reader asks, “Couldn’t you switch your body to being a fat burning machine by cutting out carbohydrates from your diet? If you only ate fat and protein, your body would have to burn the fat for energy, right?”

Good question!

Low-carbohydrate diet

And the answer is, of course, yes! The metabolic pathways while on a fast are the same processes when on a low-carbohydrate dietGCBC (Also see Eades here). This is because in both states (fat/protein-fed or fasted) your body is going to rely on free fatty acids as its main source of energy. Does it really matter that the fat comes from dietary sources or from your cells? Apparently, not3.

If you want to know a ton more about this, and I reiterate this in the footnotes, pick up Gary Taubes’ Good Calories, Bad Calories, an exhaustive book about the science and history of the low-fat-is-healthy hypothesis, the varying research studies behind low-fat diets, the research behind low-carbohydrate diets, obesity, diabetes, insulin, caloric restriction, exercising to lose weight, metabolic syndrome, some biochemistry and more. GCBC is a fascinating and eye-opening read.

As noted before, Taubes concludes that insulin is uniquely fattening. And again, since insulin is released after eating carbohydrates, carbohydrates are fattening.

Insulin is a hormone produced by your pancreas that regulates blood sugar, or glucose. Glucose comes from carbohydrates either directly or upon digestion. The presence of insulin downregulates fat mobilizing hormones (Human growth hormone, adrenaline, noradrenaline, glucagon, for example). Upon the introduction of insulin into the bloodstream, free fatty acids are driven back into fat cells for storage as they are now deemed unnecessary given the newfound energy. When all this energy leaves your bloodstream, you get hungry! In this manner, consuming carbohydrates not only shuts down fat mobilization but it can even spur hunger.

Furthermore, insulin tells your liver to take glucose and make triglycerides or fat for storage. Even more, if you note the “glyc” in triglycerides — that is for glycerol phosphate which holds the long fat chains together. Guess where your body gets glycerol phosphate? From metabolizing glucose! Thus, not only does the presence of insulin spur hunger and fat manufacturing by the liver, but the glucose facilitates fat creation by providing the molecule necessary to build the fat!

Insulin, by way of carbohydrates, is like the triple threat to being lean. It follows logically that by reducing insulin concentrations in the body, you can curb hunger, stop fat storage, and maintain the fast-induced fat mobilization process discussed earlier.

You reduce insulin concentration by fasting and avoiding carbohydrate-dense foods.

Though it is unnecessary to fast while on a low-carb / carbohydrate restricted diet, fasting speeds things along via caloric restriction overall while still allowing you to eat normal meals when you do eat. Taubes contends that a fast effectively accomplishes the same thing as Atkins’ induction phase, which is a two-week period of eating twenty grams of net carbohydrates a day or less4.

Implementation:

Though this is by no means the way to implement this diet, this is my semi-specific methodology. You must pay attention to your own results and customize a system that works for you.

Fasting: On a day when I plan to start a fast, I eat breakfast and lunch. Then I eat nothing else until dinner the next day. During the fast, I am free to drink water, tea and coffee so long as no creams or sugars are added. Some even say diet soft-drinks are ok but I universally avoid artificial sweeteners. All said, this results in anywhere from a 26 – 30 hour fast.

I make it a point to do about twenty to thirty minutes of high-intensity exercise about an hour or two prior to breaking my fast (typically some combination of multi-joint weight-lifting like dips, pull-ups, squats, kettlebell exercises, etc.). Whether you do your high-intensity exercise while in a fasted state or not, the exercise must be done! It is not optional.

I break the fast with a nutritious meal with normal-sized portions. In all meals, I avoid or eliminate carb-heavy foods. I avoid bananas and apples and all juices while regularly eating all types of berries, tomatoes and avocados. Nuts are okay though peanuts are not nuts but legumes (I still eat them from time to time though).

As far as portion size, Art DeVany says, and I think this is sound advice, to eat to satisfaction not to fullness.

Breads, potatoes, legumes and candy are off limits. Yes I cheat from time to time. I accept it and don’t allow it to overthrow my broad efforts. I find myself cheating less more and more as my body further detoxifies from the high-glucose, high-insulin addiction. I do not avoid beer or wine though I am always sure to stay hydrated and again, pay attention to your bodies!

For breakfast, I regularly eat a couple eggs (yolk never excluded), bacon or sausage (uncured) and berries. Some say avoid dairy, I do not and have not, regularly consuming cheeses and heavy cream. I do avoid milk and yogurt (can’t find any yogurt without a minimum of 15 gm of sugar). Remember: fat is ok. For me, low-carb did not mean “high protein” — it meant “high fat”. We’ve all been programmed to be scared of “high fat” — if you need some guidance in getting over your fear of fats, pick up Taubes’ book.

After breaking a fast, eat at least six meals before doing another fast.

Some notes:

While on any initial fasts, be prepared for some folks to think you are completely insane. If you keep it up, you’ll laugh when you find some of these people trying it out themselves a month later. Also, some people may experience headaches while fasting (particularly women). This is your body struggling with the swithover to fat as energy. Feel free to start with smaller fasts (say sixteen hours) before working up to a longer fast. Again, this is very personal — the nuances of insulin-sensitivity differ from person to person. Do what works for you.

If you try this out, please report any results. Questions? Comment below or email me.

Finally, a disclaimer. I am not a doctor. All of the above is for information purposes only. Any experiments you try on yourself are your own responsibility!

Conclusion:

I feel good practicing IF and a low-carbohydrate diet. The cravings for things like pizza go away faster than you might think. Excluding easy, filler-type foods makes you a smarter, more creative chef.

Via this regime, nay lifestyle, I’ve gotten back down to a body composition level akin to when I was eighteen, something I had long given up as impossible5. And I’ve done it in only a matter of a couple months.

My results being undeniable, I now have my father-in-law, wife, and brother all practicing this diet, something I now see as a way of life6 that I can maintain indefinitely. My sister-in-law and sister are also dabbling with the diet, too.

Additional Reading:

  • Fat: Mark Twight of Gym Jones talks about how endurance athletes run more efficiently on fat than on carbohydrates.
  • Martin Berkhan on Intermittent Fasting — talks about his history starting with when he was clearly overweight, to uber-skinny model, to experimenting with IF and weight lifting. Also read an interview with Martin here.
  • Fast 5 — a free ebook about fasting on a daily basis, not unlike Martin’s regiment in fast duration. A good introduction to IF.
  • Artificial Sweeteners Cause Energy Disregulation: More compelling (to me) than the arguments that artificial sweeteners cause cancer, is the contention that zero calorie sweeteners confuse our bodies, causing disregulation. I’ve avoided artificial sweeteners for the past two and a half months and haven’t missed them a bit. Note: had to link only to the clip I saved as Art has moved that post offline (or out of public view).
  • Big Fat Lie: an article from the UK Telegraph on Taubes. Read it and get the book if your interest is still piqued.
  • Intermittent Fasting: Art Devany is an expert of sorts on the paleo diet (which is low-carb by nature) and evofitness, which is high intensity “power law” training. Here he talks about his approach to IF, which of course, would have fit perfectly with the paleo lifestyle. Art is 70 and looks fantastic. Read his post on mental clarity and fasting. Note: both of these links are dead as Art redesigned his site and took much of the original content offline (unfortunately).
  • Your belly fat could be making you hungrier: A quote from the research:

    The extra fat we carry around our middle could be making us hungrier, so we eat more, which in turn leads to even more belly fat. Dr. Kaiping Yang and his colleagues at the Lawson Health Research Institute affiliated with The University of Western Ontario found abdominal fat tissue can produce a hormone that stimulates fat cell production. The researchers hope this discovery will change in the way we think about and treat abdominal obesity.

    More from Devany here. Basically, the fat in your abdomen is the last to go — almost fighting for its own existence towards the bitter end!

  • A Motivation I Haven’t Written About: Richard Nikoley talks about the link between carbohydrates and cancer cells. Apparently, cancer cells have an incredibly difficult time running on anything but glucose and even then, they burn glucose quite inefficiently. For this reason, a low-carb diet may have benefits for people fighting cancer.

There are others which I’ll have to point to in the future.

Footnotes:

GCBC Despite the name indicating the book is about foods and calories (a diet book), Good Calories, Bad Calories is a fantastic read on the history of research on low-fat and low-carb diets, insulin, weight-loss, pre-Westernized cultures and nutrition, cholesterol, cardiovascular disease, cancer (metabolic syndrome or “diseases of civilization”) and more. If you want to learn all about these topics, you must pick this book up. If you are skeptical about low-carb diets, I implore you to read GCBC. It is a fascinating read at approximately 450 pages (Note: the book is 600 pages, but 150 of them are references!).

1 Watch Taubes lecture on “Big Fat Lies” on Google video here

2 Defined as low quantity or time, high quality or work completed. Volumes could be written (and have been) on this subject. Bodyweight circuits where exercises are performed back to back (See C8B300 for an example) are one form of this. Tabata intervals are another. A Body For Life style cardio session is also popular where you do 2 minutes at Level 5 intensity, then four sessions of one minute each at levels 6, 7, 8, 9 and end with a minute at level 10 and then a minute of cooldown back at level 6 (This adds up to 20 minutes total: 2 warmup, 16 on the four minute intensity cycle, one at peak intensity, and one at cooldown).

3 Therein possibly also lies the answer to a question that arises regarding low-carb diets compared to non-low-carb caloric restriction diets. Research has shown that low-carb dieters can severely restrict calories, achieve significant weight loss, and not be hungry. Comparatively, non-low-carb calorically restricted diets wreak havoc on the dieters making them cranky and typically resulting in all the weight going back on upon ceasing the diet.

4 Many, many people I’ve talked to about this think I’m doing the Atkins diet. If I am, fine. Honestly, I haven’t read Atkins stuff well enough to say if I’m following his protocol or not. I mention the comparison of a fast and the induction phase as a means to point out the connection between fasting and low-carb diets as well as the benefit that fasting provides in terms of speeding along the process.

5 Having tried the aforeblogged Getting back to fighting weight (Post deprecated) Body for Life program and seen results, but only after untold hours in the gym (probably on the order of 100 hours) and pounding three protein shakes per day in addition to three meals.

6 Fasting twice per week is not something you have to do indefinitely. In fact, its too regular to maintain on a permanent basis. Our bodies should be kept on their toes! Periodically going to only fasting once a month would be an option (Or once every other week). Just be sure to mix it up. My guess is that once you try fasting, you’ll find that going on a periodic fast is something you want to do to clear your mind, body, etc. and/or ward off a cold, get over a weekend of gorging, whatever.

7 This diet conforms readily to the way our bodies seem to be genetically engineered by tens of thousands of years of evolution. Hunter/gatherer man wasn’t looking for french fries and rolls! It has only been in the past ten millennia that carbohydrate-dense, processed foods have been available for consumption. Our bodies simply struggle to cope with such a drastic shift in diet from nuts, berries, leafy greens (all slow-to-metabolize carbohydrates on the glycemic index), and meat to breads, rices, potatoes, and sugar which require immense amounts of insulin to regulate.